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. 2015 Mar 5;15:97. doi: 10.1186/s12885-015-1119-y

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© Ji et al.; licensee BioMed Central. 2015

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Resveratrol inhibited the morphological changes of TGF-β1-induced EMT. (A) LoVo cells were treated with 10 ng/ml TGF-β1 to induce EMT, and resveratrol with a concentration of 6, 12 μM were introduced to inhibit the morphological changes. Control LoVo cells displayed classical epithelial morphology, and 10 ng/ml TGF-β1 treated LoVo cells represented a mesenchymal phenotype. (B) Expression of epithelial phenotype marker E-cadherin and mesenchymal phenotype marker Vimentin were detected by western blot, **P < 0.01, compared with control LoVo cells without treatment of TGF-β1 and resveratrol. (C) Immunofluorescence staining of E-cadherin and Vimentin in LoVo cells treated with 10 ng/ml TGF-β1, with or without resveratrol.