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. 2015 Apr;43(4):611–619. doi: 10.1124/dmd.114.062877

Fig. 3.

Fig. 3.

Glucuronidation is impaired in tumor kidneys. Glucuronidation activity in normal and tumoral kidney homogenates for deferiprone (A), propofol (B), sorafenib (C), mycophenolic acid (D) (specifically the formation of MPA-G), and zidovudine (E). N, normal kidneys homogenates (n = 12), ○; T, tumoral kidney homogenates (n = 14), ●. Paired normal and tumoral samples are connected with a black line. Coefficients of variation (CV) among normal and tumoral samples are given. Detailed quantification data are given in Table 3. **P≤0.01; ***P≤0.001; N.S. not significant.