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. 2015 Jan 12;19(3):620–629. doi: 10.1111/jcmm.12455

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© 2015 The Authors. Journal of Cellular and Molecular Medicine published by John Wiley & Sons Ltd and Foundation for Cellular and Molecular Medicine.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Fig 1 — DHZ suppressed high-fat diet (HFD)-induced bodyweight gain. (A) Effects of DHZ on bodyweight in an HFD-induced obesity model. Data are provided in terms of means ± SD (n + 5). * Denotes significant differences compared with normal diet-fed mice (P < 0.05); *^{, #} denotes significant differences compared with HFD-fed mice (P < 0.05). (B) Effects of DHZ on visceral fat in an HFD-induced obesity model. (C) Effects of DHZ on fatty liver in an HFD-induced obesity model. (D) Effects of DHZ on the kidney and peri-renal fat in an HFD-induced obesity model. (E) Effects of DHZ on the epididymal fat in an HFD-induced obesity model. (F) Effects of DHZ on the size of adipocyte of an HFD-induced obesity model. (G) Effects of DHZ on the weight of liver and epididymal fat tissues of an HFD-induced obesity model. The three groups were as follows: LFD, low-fat diet; HFD, high-fat diet and HFD supplemented with 100 mg/kg/day DHZ.