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. 2015 Mar 27;9:78. doi: 10.3389/fnbeh.2015.00078

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Copyright © 2015 Manto, Honnorat, Hampe, Guerra-Narbona, López-Ramos, Delgado-García, Saitow, Suzuki, Yanagawa, Mizusawa and Mitoma.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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GAD65Ab titers and epitope specificities in patients with neurological disorders. GAD65Ab titers (A) and epitope specificities (B,C) in patients with SPS, cerebellar ataxia, SPS, LE, and type 1 diabetes patients without neurological symptoms were evaluated. Results for the LE patient that presents also stiff-leg syndrome are indicated as an open circle. GAD65Ab titers are presented as U/ml. Binding to GAD65 in the presence of rFab is presented as percent related to non-competed binding (set as 100%). Median binding is indicated. Significant differences are shown. Cut-off value for significant competition of GAD65Ab binding by recombinant Fab is indicated as the dotted line in panels (B,C). * = P < 0.05, ** = P < 0.01.