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a Proposed pathogenesis of type 1 and type 2 diabetes as related to the differential timelines of their associated CVD complications. HLA human leucocyte antigen, AAb autoantibody. b Schematic of the pathogenesis of post-infarction autoimmunity (PIA) in T1D patients and humanized DQ8+NOD mice and how PIA is avoided in non-autoimmune-prone (“control”) T2D patients and non-autoimmune-prone B6 mice. NOD non-obese diabetic, B6 C57BL/6, α-MyHC α-isoform of cardiac myosin heavy chain
