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TMP and NAC inhibited arsenic-induced pro-inflammatory signals: NF-κB activation and β-catenin up-regulation in HK-2 cells. a, b Arsenic-induced NF-κB activation was determined by increased levels of nuclear phospho-Ser536 NF-κB p65, which was inhibited by NAC or TMP pretreatment. c, d Arsenic-induced β-catenin upregulation was suppressed by NAC and TMP pretreatment. Histone H3 (Histon) and β-actin were used as loading controls for nuclear and total proteins, respectively
