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. Author manuscript; available in PMC: 2015 Oct 1.
Published in final edited form as: J Antibiot (Tokyo). 2014 Oct 1;68(4):271–278. doi: 10.1038/ja.2014.133

Table 4.

Potential targets for non-replicating M. tuberculosis.

  • Electron transport systems

  • Menaquinone biosynthesis

  • FoF1 ATP synthase

  • Activation via reductase(s) to form cytotoxic species.

  • Protein kinase (e.g. PknA, PknB, and PknG)

  • DosS/DosR systems

  • Glyoxylate pathway

  • Methyltransferase

  • Protein biosynthesis

  • DNA gyrase