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. 2015 Feb 25;8:124. doi: 10.1186/s13071-015-0715-7

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© Alvarez Rojas; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Change in transcription of genes involved in hepatotoxicity (blue bars), fibrosis (orange) and extracellular matrix formation (ECM) (red). Legend: Names on vertical axis represent proteins encoded by these genes. Coloured bars indicate the fold change between TPM values for infected compared with control sheep. Fold-change on the horizontal axis was calculated using averaged TPM values for infected and control groups. TGLN: transgelin, SLC2A3: solute carrier family 2 member 3, TIMP1: tissue inhibitor of metalloproteinases 1, TGF-β1: transforming growth factor beta 1, TNF: tumour necrosis factor, COL1A1: collagen, type I, alpha 1, OST: osteopontin and COL16A1: collagen, type I, alpha 1: collagen, type XVI, alpha 1.