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Schematic of SO2 cellular mechanisms. Effects of leukocyte recruitment in the airway following SO2 exposure, as well as effects of SO2 itself, are shown. ROS, as a direct product from SO2 exposure or via secretion from recruited leukocytes 1) promotes an oxidant status shift within the epithelial cell and 2) modulates gene and protein levels, which feed back into the oxidant status shift within the epithelial cell. X is the site of possible IL-10 inhibitory effects.
