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Chronic nicotine treatment enhances aortic smooth muscle relaxation in a time-dependent manner. Dose-response curves of SNP-induced relaxation in aortas pre-contracted with a single dose of Phe (3×10−7 mol/L) after 1 week, 3 weeks and 6 weeks nicotine treatment in Sprague-Dawley rats. Sal, saline group; Nic, nicotine group; F−E−, both adventitial fat and endothelium removed. Data are shown as mean±SEM. n=4–8. cP<0.01.
