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. 2015 Jan 27;3(1):e12278. doi: 10.14814/phy2.12278

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© 2015 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Figure 7. — Endogenous FGF2 mediates capillary remodeling after cardiac ischemia‐reperfusion injury. (A) Representative images of capillaries in Fgf2^−/− and wild‐type mice at 7 days after IR injury or sham treatment (red – CD31, blue – DAPI). (B) Fgf2^−/− mice have decreased numbers of capillaries (normalized to number of cells present) in the peri‐infarct area at 3 and 7 days after IR injury compared to wild‐type controls. (C) There is no significant difference in capillary size in the peri‐infarct area in any treatment group. n = 3–8, *P < 0.05 versus wild type, ^#P < 0.05 versus sham, ⁺P < 0.05 versus 1 day time point.