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. 2015 Apr 9;10(4):e0123776. doi: 10.1371/journal.pone.0123776

Fig 2. Somite patterning and vertebral column defects in single and double Dll3 and Lfng mutants.

Fig 2

Whole-mount in situ hybridizations of E9.5 embryos with an Uncx4.1 specific probe (a-d) and lateral (e-h) and dorsal (e‘-h‘) views of skeletal preparations of wild type (a, e,e‘), homozygous Dll3 pu (b, f, f‘), homozygous Lfng lacZ (c, g, g‘) and double homozygous Dll3 pu; Lfng lacZ E18.5 embryos (d, h, h‘). Absence of both proteins does not enhance somite A-P patterning or vertebral column defects present in single mutants.