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. Author manuscript; available in PMC: 2015 Apr 15.
Published in final edited form as: Free Radic Biol Med. 2010 Jul 13;49(6):1119–1128. doi: 10.1016/j.freeradbiomed.2010.06.030

Fig. 1.

Fig. 1

The cytoplasmic tail of ICAM-1 increases production of GSH in a time-dependent manner. (A) The amino acid sequences of the AP and AP-ICAM peptides. (B) GSH levels after cellular treatment with various concentrations of the cytoplasmic tail peptide AP-ICAM. (C) GSH levels measured at various time points after cellular treatment with AP or AP-ICAM peptide (normalized to vehicle). (D) GSH levels after treatment with the AP-ICAM peptide plus BSO (normalized to AP-ICAM treatment alone). n=5; #p<0.001, + p<0.05; experiments performed in quadruplicate.