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. Author manuscript; available in PMC: 2015 Apr 15.
Published in final edited form as: Free Radic Biol Med. 2010 Jul 13;49(6):1119–1128. doi: 10.1016/j.freeradbiomed.2010.06.030

Fig. 8.

Fig. 8

AP-ICAM peptide blocks VEGF-dependent chemotaxis in a glutathione-dependent manner. (A) The percentage of endothelial cell transmigration over time in response to VEGF-A (50 ng/ml), VEGF-A plus AP (25 µM) peptide, or VEGF-A plus AP-ICAM (25 µM) peptide. (B) The effect of BSO treatment (150 µM) on VEGF-A chemotaxis with or without peptide treatment. n=5; #p<0.001, *p<0.01; experiments performed in triplicate.