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. 2015 Mar 16;43(7):3546–3562. doi: 10.1093/nar/gkv211

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© The Author(s) 2015. Published by Oxford University Press on behalf of Nucleic Acids Research.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 6. — Additional subunits of the SAGA complex show genetic interaction with invadolysin, in an invadolysin-induced rough-eye modification assay. Images show Drosophila compound eyes. (A) A rough-eye phenotype results when the expression of UAS-invadolysin is induced in the eye by a GMR-GAL4 driver (compare with the wild-type eye). (B) The invadolysin-induced rough-eye phenotype is suppressed by mutations of subunits of both DUB (not and sgf11) and HAT (gcn5 and ada2b) modules of the SAGA complex.