Table 1. A second site non-complementation screen to identify genetic interactors of invadolysin.
| Df screen | Cytology | Interaction | Df screen | Cytology | Interaction |
|---|---|---|---|---|---|
| Df(1)ED404 | 1D2; 1E3 | − | Df(3L)ED212 | 65A9; 65D5 | - |
| Df(1)ED6630 | 3B1; 3C5 | − | Df(3L)ED4408 | 66A22; 66C5 | - |
| Df(1)ED418 | 5C7; 5E4 | − | Df(3L)ED4421 | 66D12; 67B3 | - |
| Df(1)ED6957 | 8B6; 8C13 | − | Df(3L)ED4475 | 68C13; 69B4 | - |
| Df(1)ED429 | 9D3; 9D3 | − | Df(3L)ED4483 | 69A5; 69D3 | - |
| Df(1)ED7161 | 11A1; 11B14 | − | Df(3L)ED215 | 69B5; 69C4 | - |
| Df(1)ED7217 | 12A9; 12C6 | − | Df(3L)ED4486 | 69C4; 69F6 | - |
| Df(1)ED7294 | 13B1; 13C3 | − | Df(3L)ED4502 | 70A3; 70C10 | - |
| Df(1)ED447 | 17C1; 17F1 | − | Df(3L)ED217 | 70F4; 71E1 | - |
| Df(1)ED7413 | 17D1; 17F1 | − | Df(3L)ED218 | 71B1; 71E1 | - |
| Df(1)ED7441 | 18A3; 18C2 | − | Df(3L)ED219 | 71E1; 72F1 | - |
| Df(2L)ED2809 | 21B1; 21B1 | − | Df(3L)ED220 | 72D4; 72F1 | - |
| Df(2L)ED5878 | 21B1; 21B3 | − | Df(3L)ED223 | 73A1; 73D5 | - |
| Df(2L)ED62 | 21D1; 21E2 | − | Df(3L)ED4674 | 73B5; 73E5 | - |
| Df(2L)ED87 | 21E2; 21E2 | − | Df(3L)ED4685 | 73D5; 74E2 | - |
| Df(2L)ED123 | 22B8; 22D4 | − | Df(3L)ED4710 | 74D1; 75B11 | - |
| Df(2L)ED284 | 25F2; 26A3 | − | Df(3L)ED224 | 75B1; 75C6 | - |
| Df(2L)ED508 | 28B1; 28C4 | − | Df(3L)ED225 | 75C1; 75D4 | + |
| Df(2L)ED606 | 28E1; 29C3 | − | Df(3L)ED4782 | 75F2; 76A1 | - |
| Df(2L)ED629 | 29B4; 29E4 | − | Df(3L)ED4786 | 75F7; 76A5 | - |
| Df(2L)ED623 | 29C1; 29E4 | − | Df(3L)ED4799 | 76A1; 76B3 | - |
| Df(2L)ED647 | 29E1; 29F5 | − | Df(3L)ED228 | 76A1; 76D2 | - |
| Df(2L)ED695 | 30C5; 30E4 | +/− | Df(3L)ED229 | 76A1; 76E1 | - |
| Df(2L)ED784 | 34A4; 34B6 | − | Df(3L)ED4858 | 76D3; 77C1 | - |
| Df(2L)ED3 | 35B2; 35D1 | − | Df(3L)ED4861 | 76F1; 77E6 | - |
| Df(2L)ED1050 | 35B8; 35D4 | − | Df(3L)ED4978 | 78D5; 79A2 | - |
| Df(2L)ED1109 | 36A3; 36A10 | − | Df(3L)ED230 | 79C2; 80A4 | - |
| Df(2L)ED1203 | 36F7; 37C5 | − | Df(3L)ED5017 | 80A4; 80C2 | - |
| Df(2L)ED1303 | 37E5; 38C6 | − | Df(3L)ED231 | 80B1; 80C1 | - |
| Df(2R)ED1552 | 42A11; 42C7 | +/− | Df(3R)ED5071 | 82A1; 82E4 | +/- |
| Df(2R)ED1612 | 42A13; 42E6 | − | Df(3R)ED5138 | 82D5; 82F8 | - |
| Df(2R)ED1618 | 42C3; 43A1 | − | Df(3R)ED5156 | 82F8; 83A4 | - |
| Df(2R)ED1715 | 43A4; 43F1 | − | Df(3R)ED5177 | 83B4; 83B6 | - |
| Df(2R)ED1725 | 43E4; 44B5 | − | Df(3R)ED5196 | 83B9; 83D2 | - |
| Df(2R)ED1791 | 44F7; 45F1 | − | Df(3R)ED5230 | 84E6; 85A5 | - |
| Df(2R)ED2219 | 47D6; 48B6 | − | Df(3R)ED5412 | 85D15; 85E6 | - |
| Df(2R)ED2426 | 51E2; 52B1 | − | Df(3R)ED5514 | 86C7; 86E11 | - |
| Df(2R)ED2736 | 53C6; 53F8 | − | Df(3R)ED5516 | 86D8; 86E13 | - |
| Df(2R)ED3610 | 54F1; 55C8 | − | Df(3R)ED5577 | 86F9; 87B13 | - |
| Df(2R)ED3683 | 55C2; 56C4 | − | Df(3R)ED5591 | 87B7; 87C7 | - |
| Df(2R)ED3923 | 57F6; 57F10 | − | Df(3R)ED5610 | 87B11; 87D7 | - |
| Df(2R)ED4071 | 60C8; 60E8 | − | Df(3R)ED5622 | 87F10; 88A4 | - |
| Df(3L)ED4079 | 61A5; 61B1 | − | Df(3R)ED5644 | 88A4; 88C9 | - |
| Df(3L)ED201 | 61B1; 61C1 | − | Df(3R)ED5705 | 88E12; 89A5 | - |
| Df(3L)ED4177 | 61C1; 61E2 | − | Df(3R)ED5780 | 89E11; 90C1 | - |
| Df(3L)ED4191 | 61C3; 62A2 | − | Df(3R)ED2 | 91A5; 91F1 | - |
| Df(3L)ED207 | 61C9; 62A6 | − | Df(3R)ED5911 | 91C5; 91F8 | - |
| Df(3L)ED4284 | 62B4; 62B12 | − | Df(3R)ED5942 | 91F12; 92B3 | - |
| Df(3L)ED4287 | 62B4; 62E5 | − | Df(3R)ED6076 | 93E10; 94A1 | - |
| Df(3L)ED4288 | 63A6; 63B7 | − | Df(3R)ED6096 | 94B5; 94E7 | - |
| Df(3L)ED208 | 63C1; 63F5 | − | Df(3R)ED6220 | 96A7; 96C3 | - |
| Df(3L)ED4344 | 63F5; 64B12 | − | Df(3R)ED6232 | 96F10; 97D2 | - |
| Df(3L)ED4342 | 64A12; 64B12 | − | Df(3R)ED6235 | 97B9; 97D12 | - |
| Df(3L)ED210 | 64B9; 64C13 | − | Df(3R)ED6265 | 97E2; 98A7 | - |
| Df(3L)ED211 | 65A9; 65B4 | − | Df(3R)ED6310 | 98F12; 99B2 | - |
IX-141 mutants balanced over TM3 or TM6B were crossed to DrosDel deficiencies (e.g. Df) balanced over an appropriate balancer depending on the chromosome. The progeny were scored for the presence or absence of balancers (using dominant markers phenotypes associated with balancers). The transheterozygous flies with no balancer could be viable, semi-lethal or lethal based on the interaction between IX-14 and genes deleted within the deficiency tested.
−: no interaction.
+/−: weak interaction, semi-lethal transheterozygous progeny.
+: strong interaction, lethal transheterozygous progeny.