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. 2015 Apr 22;6:328. doi: 10.3389/fmicb.2015.00328

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Copyright © 2015 Kurth, Belfiore, Gorriti, Cortez, Farias and Albarracín.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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UV-resistome in Acinetobacter Ver3. Proteins with experimental evidence in Ver3 supporting its UV-protective function are in bold. DNA repair systems: photolyase has an important role in repairing DNA damage. Oxidative stress resistance: catalase is an inducible protein with a major role in defense against the oxidative stress. Tolerance to damage: Acinetobacter sp. Ver3 is able to resist damage in its DNA without losing viability. This might be due to error prone polymerases. Regulation: different systems are involved in resistance. Regulation may or may not be coordinated. Several regulators were identified; one is slightly induced while growing the strain in UV. However, none seems to directly detect UV. Regulation would respond to other signals.