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. 2015 Apr 22;3:55. doi: 10.3389/fbioe.2015.00055

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Copyright © 2015 Angelova, Geletneky, Nüesch and Rommelaere.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Characteristics of H-1 parvovirus. (A) In silico model of H-1PV capsid surface showing the two, three, and fivefold axes of symmetry (Allaume et al., 2012). (B) Simplified viral gene expression map. The viral single-stranded (ss) DNA genome ends in unique palindromic sequences (Pal), which serve as self-priming origins of replication for the synthesis of double-stranded replication forms/transcription templates. Transcription is controlled by the P4 and P38 promoters that are indicated by arrows and direct expression of non-structural (NS) and capsid (VP) proteins, respectively. (C) Simplified scheme of the viral life-cycle depicting the main steps leading to virus production and induction of cytopathic effects (CPE). Known and putative oncogenic transformation-stimulated steps are indicated by full and dashed red circles, respectively.