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. Author manuscript; available in PMC: 2015 Apr 24.
Published in final edited form as: Dev Biol. 2009 Jan 23;328(2):234–244. doi: 10.1016/j.ydbio.2009.01.017

Table 2.

Rescue of dsh-2(or302) hypodermal morphogenesis defects with dsh-2 deletion constructs

Array Dorsal intercalation defectivea Total embryonic lethalityb Ventral ruptureb Body shape defectsb
dsh-2(or302) 100% (n=38) 86.3% (n=51) 58.8% (n=51) 80.8% (n=30)e
Full length graphic file with name nihms680804t1.jpg 6.1% (n=33) 14.9% (n=108) 5.6% (n=108) 14.9% (n=103)
ΔDIX graphic file with name nihms680804t2.jpg 57.1% (n=35)c 29.4% (n=102) 12.1% (n=102) 31% (n=87)
ΔPDZ graphic file with name nihms680804t3.jpg 68.6% (n=35)c,d 46% (n=87) 20.7% (n=87) 78.9% (n=71)e
ΔDEP graphic file with name nihms680804t4.jpg 81.5% (n=27)d 59.5% (n=126) 31.7% (n=126) 58.8% (n=68)

DSH-2 domains that are present in the constructs are as indicated.

a

Presence of intercalation defects as analyzed using both Nomarski imaging and confocal imaging with the junctional marker AJM-1::GFP.

b

Embryonic lethality, ventral rupture, and body shape defects observed in Nomarski movies of developing embryos.

c

Not significantly different, p=0.12.

d

Not significantly different, p=0.20.

e

Not significantly different, p=0.56.