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. 2015 Mar 27;42(4):344–352. doi: 10.1111/1440-1681.12360

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© 2015 The Authors. Clinical and Experimental Pharmacology and Physiology published by Wiley Publishing Asia Pty Ltd.

This is an open access article under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Fig 3 — Plasma noradrenaline levels (Aa), plasma renin activity (PRA) (b), and plasma angiotensin I (Ang I) levels (c) in spontaneously-hypertensive rats (SHR) treated with adriamycin (ADR), followed by 4 weeks of vehicle (ADR group), 20 mg/kg cilnidipine (ADR-Cil), or 3 mg/kg amlodipine (ADR-Aml) administration. Saline group received saline rather than ADR, followed by vehicle for 4 weeks. Values are the mean ± standard error of the mean. ^†††P < 0.001 for the comparison with the saline group; *P < 0.05, ***P < 0.001 for the comparison with the ADR group; ^§§§P < 0.001 for the comparison with the ADR-Aml group. , Saline; , ADR; , ADR-Cil; , ADR-Aml.

Inline graphic — Plasma noradrenaline levels (Aa), plasma renin activity (PRA) (b), and plasma angiotensin I (Ang I) levels (c) in spontaneously-hypertensive rats (SHR) treated with adriamycin (ADR), followed by 4 weeks of vehicle (ADR group), 20 mg/kg cilnidipine (ADR-Cil), or 3 mg/kg amlodipine (ADR-Aml) administration. Saline group received saline rather than ADR, followed by vehicle for 4 weeks. Values are the mean ± standard error of the mean. ^†††P < 0.001 for the comparison with the saline group; *P < 0.05, ***P < 0.001 for the comparison with the ADR group; ^§§§P < 0.001 for the comparison with the ADR-Aml group. , Saline; , ADR; , ADR-Cil; , ADR-Aml.