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. 2015 Apr 22;15:125. doi: 10.1186/s12906-015-0652-1

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© Lee et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Effects of F. mume on chronic BCCAo-induced astrocytic activation in the white matter. Immunohistological staining was conducted to investigate the expression of GFAP-positive cells in the white matter and the hippocampus in the sham-operated group (n = 7-8), the BCCAo + Vehicle group (n = 9), and the BCCAo + F. mume group (n = 9). Representative photomicrograph of GFAP (A and B) positive cells. The number of GFAP (C and D)-positive cells was increased in the cerebral cortex, fimbria, the optic tract and CA1 of the BCCAo-injured rats compared to the sham-operated rats (*), and was significantly decreased in the optic tract of the BCCAo rats treated with F. mume compared to the BCCAo-injured rats (#). Data were analyzed via ANOVA followed by the Tukey test. *, p < 0.05 versus the BCCAo + Vehicle group; #, p < 0.05 versus the BCCAo + F. mume group. CA 1 and 3, cornuammonis 1 and 3; DG, dentate gyrus.