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. 2015 May 1;10(5):e0125903. doi: 10.1371/journal.pone.0125903

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© 2015 Wang et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 6 — P4 activated cSrc, subsequently causing membrane translocation of Kras, which in turn activated Raf-1/AKT/ERK1/2/p38/IκBα and increased nuclear translocation of NFκB (p65), and eventually increasing the levels of p21^cip1 and p27^kip1 protein through up-regulating p53 expression in RASMCs. PR, progesterone receptor.