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. 2015 May 4;10(5):e0125692. doi: 10.1371/journal.pone.0125692

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© 2015 Muñoz-González et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 6 — (a) Expression of 5 different surface markers and CSFV in BMHCs from infected and non-infected pigs. (b) Comparative phenotypes of BMHCs obtained from non-infected pigs (i, iv) and pigs infected with the PR (ii, v) and Cat01 (iii, vi) strains. (i, ii and iii): forward scatter (relative cell size, x-axis) and side scatter (relative granularity, y-axis). (iv, v and vi): double labelling immunofluorescence image of the BMHCs from (i, ii and iii), in terms of CD172a common myeloid marker (y-axis) and SLA-II (x-axis). (c) Percentage of granulocyte 6D10⁺ and 6D10⁺_CSFV⁺ double-positive cells in the BMHCs. (d) Double labelling immunofluorescence image of the BMHCs from one non-infected and two infected pigs, in terms of 6D10, immature granulocytes marker (x-axis) and CSFV (y-axis). (e) Sorting of 6D10⁺ BMHCs. These experiments were repeated twice under the same conditions.