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. 2015 Apr 14;17(1):100. doi: 10.1186/s13075-015-0619-0

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© Zong et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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GPI modulates the secretion of TNF-α, IL-1β, TGF-β and IL-6 in FLS isolated from RA or OA patients. RA or OA FLS were transfected with GPI expression plasmids and siRNAs or treated with GPI at 1 μg/ml for 72 hours and the supernatants were collected for the analysis of TNF-α (A and B), IL-1β (C and D), TGF-β (E and F) and IL-6 (G and H) levels using ELISA. Data were shown as mean ± SD (N = 8, R = 3). P values were determined by Wilcoxon signed-rank test. (N = number of individuals, R = number of replicates).