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. 2015 Apr 20;36(5):572–586. doi: 10.1038/aps.2014.159

Figure 6.

Figure 6

Prolactin (PRL) induced endothelial- and phenylephrine (Phe)-mediated vascular effects in isolated rat aortic rings. Vessels were precontracted with 2 μmol/L Phe. Representative trace recordings of tension, in grams (g), in response to (A) cumulative (0.01–100 nmol/L) concentrations of PRL were administered in the presence of 2 μmol/L Phe −E; (B) cumulative (0.01–100 nmol/L) concentrations of PRL were administered in the absence of 2 μmol/L Phe 2 +E; (C) a single dose of 0.01 nmol/L PRL was administered in the presence of 2 μmol/L Phe +E; (D) the percentage of relaxation (% of contraction in response to Phe) in the aortic rings following the administration of single doses of PRL (0.01–100 nmol/L). Values are mean±SEM. n=5 animals in each group. Data that were not statically significant, bP<0.05. +E, in the presence of endothelium, −E, in the absence of endothelium.