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. Author manuscript; available in PMC: 2016 Jul 1.
Published in final edited form as: Behav Brain Res. 2015 Mar 21;287:89–95. doi: 10.1016/j.bbr.2015.03.023

Figure 1. FST phenotype of loss-of-function NMDAR, AMPAR and PSD-95 mutations.

Figure 1

(A) GluN1INTER-KO mutants showed similar immobility to floxed controls (n=7–18 per genotype). (B) GluA1KO mutants showed less immobility than WT controls (n=7–8). (C) PSD-95 mutants showed significantly less than WT controls (n=10–11 per genotype). Data are means ± SEM. *P<.05 WT versus KO