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. 2015 May 6;16(1):54. doi: 10.1186/s12931-015-0214-6

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© Sheridan et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Basal miR-146a expression in human lung fibroblasts from Normal subjects as well as smokers with and without COPD. (A) miR-146a-lung tissue: There was a significant increase in miR-146a in lung tissue derived from COPD subjects compared to Normal. Results are expressed as mean ± SEM and each symbol represents a different individual (Normal, n = 10; At Risk, n = 9; COPD = 9). (B) miR-146a-fibroblasts: There was no significant difference in basal miR-146a expression in lung fibroblasts derived from the three patient groups. Results are expressed as the mean ± SEM of miR-146a levels normalized to the snRNA U6 and are expressed as fold-change compared to Normal fibroblasts. Each symbol represents fibroblasts from a different individual (Normal, n = 5; At Risk, n = 13; COPD = 11).