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. Author manuscript; available in PMC: 2015 May 17.
Published in final edited form as: Kidney Int. 2010 Jul 7;78(7):650–659. doi: 10.1038/ki.2010.197

Figure 7. Alterations in abundance of β-ENaC and ANP signaling components in proteinuric kidney diseases.

Figure 7

Western blots of medullary β-ENaC, PDE5, phospho-PDE5 (p-PDE5), PKGII and β-actin (as loading control). Compared to controls strongly increased expression levels are present in PAN (grey bar) and GN (white bar) rats for all parameters analyzed. Densitometric evaluations are presented below. Values are means ± SE; n=5 per group; *P < 0.05.