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. 2015 May 18;7:7. doi: 10.3389/fnsyn.2015.00007

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Copyright © 2015 Stincic and Frerking.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution and reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Blockade of EAAT1 recruits extrasynaptic AMPARs that predominantly contain GluA2. (A) Representative traces of before (black) and after addition of TBOA (green). TBOA results in a small reduction of the fast component and a major enhancement of the tail current. (B) Representative traces of the EPSC in TBOA, before (green) and after (red) PhTx. (C) Group average PhTx-resistant (red) and—sensitive (blue) currents are shown normalized to total response in TBOA. The PhTx-sensitive current is only a small portion of the total current and does not preferentially contribute to the tail. (D) Group average PhTx-resistant (red) and—sensitive (blue) currents are shown normalized to their own total response in order to compare kinetics. A bi-exponential fit found no significant differences for either the relative proportions of the current or the time constants of the two currents.