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Effect of ATK on CEC-induced locomotor deficits. Animals were treated with ATK orally at 2 h following CEC, and the same dose was repeated daily until the end point (14 days). Motor function was evaluated by rotarod task on indicted days. Sham-operated animal had normal motor function (latency 269 ± 19 s). Data are presented as mean ± SD (n = 8). **p < 0.01, ***p < 0.001 vs. CEC + VEH. Neither group reached sham levels over the testing period (p < 0.001).
