Table 2.
Interaction models of CYP1A1 and GSTM1 polymorphism with former smoker in intestinal gastric cancer
| Genotype | Former smoker | Cases | Controls | OReg | 95%CI |
| Ile1 | No | 57 | 391 | 1 | Ref. |
| Val2 | No | 29 | 254 | 0.7 | 0.4–1.1 |
| Ile | Yes | 13 | 21 | 1.8 | 0.7–4.3 |
| Val | Yes | 13 | 10 | 5.05 | 1.9–13.4 |
| Present3 | No | 31 | 302 | 1 | Ref. |
| Null4 | No | 54 | 345 | 1.9 | 1.1–3.1 |
| Present | Yes | 10 | 12 | 3.5 | 1.2–10.1 |
| Null | Yes | 17 | 16 | 8.46 | 3.4–20.9 |
1
[Ile]: CYP1A1 homozygous (Ile/Ile) genotype;
2
[Val]: CYP1A1 heterozygous (Ile/Val)+homozygous (Val/Val) genotypes;3[present]: present genotype for GSTM1;
4
[null]: null genotype for GSTM1;
5
adjusted by age, gender, living areas, family history of cancer, and former alcohol drinking; χ2trend=24.0, df=1, P=0.00; γ=1.61/0.57=2.8;
6
adjusted by age, gender, living areas, family history of cancer, and former alcohol drinking; χ2trend=45.7, df=1, P=0.00; γ=2.13/0.63=3.4.