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. 2015 May 21;10(5):e0127434. doi: 10.1371/journal.pone.0127434

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© 2015 Karimian et al

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.

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Fig 6 — Tumor control (A), FALHE low-dose treatment (B), FALHE high-dose treatment (C), and tamoxifen treatment (D). Dark brown particles are representing the expression of Bax, Bcl-2, Caspase 3 and p53 proteins. Microscopic observation of the FALHE-treated group compared with the control tumor showing high expression levels of Bax, p53 and caspase 3 and a low expression level of Bcl-2 protein. This result demonstrates the activation of the intrinsic pathway of apoptosis. Magnification, 40×.