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. 2015 May 20;34(1):52. doi: 10.1186/s13046-015-0172-3

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© Li et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Effect of GC-MSCs on gastric cancer cell proliferation assessed by CFU assay. (A) Representative images of BGC-823 cell colony formation treated with 10 % CM from GC-MSCs, GCN-MSCs, or BM-MSCs. (B) Average colony numbers ± SD were plotted of BGC-823 cells in different groups (n = 3). (C) Representative images of MKN-28 cell colony formation treated with 10 % CM from GC-MSCs, GCN-MSCs, or BM-MSCs. (D) Average colony numbers ± SD were plotted of MKN-28 cells in different groups (n = 3). *P < 0.05: compared with the control group; ^# P < 0.05: compared with the GC-MSC-CM treated group