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. 2015 May 12;2015:426379. doi: 10.1155/2015/426379

Table 2.

Pathogenic CNVs and variants of unknown significance detected by chromosomal microarray analysis (CMA) in fetuses with CNS anomalies.

Case number GA CNS anomalies Associated anomalies CMA result Size (Mb) CNV type OMIM or corresponding disorder
1 23+ Holoprosencephaly, single nostril No arr7q36.
(155,473,296–158,909,738) × 1
3.44 Loss SHH gene (600725)

2 22+ Hydrocephaly No arr22q11.21
(18,648,855–21,800,471) × 3
3.15 Gain 22q11.2 duplication syndrome (#608363)

3 25+ Dandy-Walker syndrome VSD
PLSVC
arr2q13q14.1
(111,596,906–114,844,660) × 1
3.25 Loss PAX8 (167415)
IL1B (147720)
MERTK (604705)
IL1RN (147679)

4 23+ Dandy-Walker syndrome Skeletal dysplasia
lip and palate cleft
absence of septum pellucidum
arachnoid cyst
arrXq13.3
(74,171,888–74,343,340) × 1
0.17 Loss ABC7 (300135)

5 23+ Holoprosencephaly Lip and palate cleft arr2p21
(44,749,075–45,098,283) × 1
0.34 Loss SIX 3 gene (603714)

6 21+ Exencephaly No arr19p12p13.11
(19,838,485–23,868,512) × 1
4.03 Loss

7 24+ Holoprosencephaly VSD
facial anomaly
arr4q35.2
(187,979,723–190,767,114) × 1
2.79 Loss

8 25+ Hydrocephaly Thickened NF
IUGR
sacrococcygeal vertebral anomaly
arr 21q21.1
(22,508,434–23,663,338) × 1
1.15 Loss

GA, gestational weeks; VSD, ventricular septal defect; PLSVC, persistent left superior vena cava; NF, nuchal fold; IUGR, intrauterine growth retardation.

Human genome build was hg19.