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. 2015 May 1;19(1):200. doi: 10.1186/s13054-015-0919-4

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© Li et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Delayed kallistatin treatment reduces renal injury and decreases blood urea nitrogen (BUN), serum creatinine, interleukin-6 (IL-6) and high mobility group box-1 (HMGB1) levels in cecal ligation and puncture (CLP) mice. (A) H&E staining was performed on kidney sections to examine renal histology (n = 4). The representative sections are shown at ×200 magnification. Levels of (B) BUN, (C) serum creatinine, (D) IL-6 and (E) HMGB1 were significantly decreased by kallistatin administration in CLP mice (n = 3 to 4). Data are expressed as means ± SE. *P <0.05 versus sham group; ^# P <0.05 versus CLP control group.