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. 2015 May 1;19(1):200. doi: 10.1186/s13054-015-0919-4

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© Li et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Delayed kallistatin administration attenuated liver injury, serum alanine transaminase (ALT) levels and liver tumor necrosis factor-α (TNF-α) expression in cecal ligation and puncture (CLP) mice. (A) H&E staining was performed on liver sections to examine liver histology (n = 4). Representative sections are shown at ×200 magnification. (B) Serum ALT levels and (C) liver TNF-α expression were significantly decreased in kallistatin-treated CLP mice (n = 3 to 4). Data are expressed as means ± SE. *P <0.05 versus sham group; ^# P <0.05 versus CLP control group.