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. Author manuscript; available in PMC: 2016 Jun 1.
Published in final edited form as: J Neuroendocrinol. 2015 Jun;27(6):487–497. doi: 10.1111/jne.12252

Fig. 5.

Fig. 5

Dendritically-released vasopressin (VP) contributes to osmotieally-driven renal sympathetic nerve activity (RSNA). (a) Recordings or RSNA after intracar-otid infusions of an isosmotic (NaCl 0.3 osmol/l) or hyperosmotic (NaCl 2.1 osmol/l) solution, in the absence or presence of bilateral microinjections of the V1a receptor antagonist (0.4 mg/ml) into the paraventricular nucleus (PVN). (b) Summary data showing a dose-dependent increase of RSNA after intracarotid infusions of NaCI. Note the blunted sympathetic response after an intra-PVN microinjection of the V1a receptor antagonist [*P < 0.0001 versus respective artificial cerobrospinal fluid (aCSF)]. Modified from Brussaard et al. (56).