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GM-CSF enhanced the association between VE-cadherin and VEGFR2 in burn serum conditions, and this effect was not observed in RhoA-overexpressing cells (A). RhoA overexpression decreased pAkt expression, even with GM-CSF treatment (C). RhoA overexpression attenuated the GM-CSF functions in the reduction of cell apoptosis and increased cell viability after burn serum damaged (C, D) (*p < 0.05).
