Fig. 9.

A model representing the effects of salt stress on the wild type and jasmonate biosynthesis mutants under salt stress. The observed phenotype of increased salt sensitivity in the wild type could be linked to the accumulation of higher amounts of MDA and OPDA as a result of higher lipid peroxidation due to ROS accumulation. Both MDA and OPDA are thought to destroy PSII and induce GSH depletion, resulting in disarmed antioxidative ability in the wild type. The mutants showed less accumulation of OPDA and MDA under salt stress conditions and less necrotic symptoms.