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. Author manuscript; available in PMC: 2015 Jun 5.
Published in final edited form as: J Glycomics Lipidomics. 2010 Nov 18;1(1):101.

Figure 1.

Figure 1

Assessment of neurological deficit scores and histochemistry confirm that NAE 16:0 protects against ischemic brain damage following MCAO I/R.

(A) MCAO-mediated I/R caused moderate to severe neurological deficits, as measured with the standardized neurological deficit score, and treatment with NAE 16:0 reduced neurological deficits significantly (phenotype: none to mild). (B) The size of the ipsilateral ischemic lesion volume (infarct volume) was significantly reduced by NAE 16:0 treatment. Data are shown as mean ± SE. a P < 0.05, compared with I/R vehicle-treated group as determined using one-way ANOVA with Newman Keuls multiple comparison post-hoc test.