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. Author manuscript; available in PMC: 2015 Dec 1.
Published in final edited form as: Nat Med. 2015 May 11;21(6):591–600. doi: 10.1038/nm.3856

Figure 2. gMDSC are transiently induced in acute HBV infection and decline before acute and chronic hepatic flares.

Figure 2

gMDSC were quantitated as a percentage of total myeloid cells (CD11bhighCD33+) from PBMC cryopreserved from multiple time points from the onset of viraemia and subsequent hepatic flare. a) gMDSC frequencies in relation to viral load in three subjects with acute HBV infection sampled from the pre-clinical phase through to HBV disease resolution. b) gMDSC frequencies plotted with the temporal course of liver inflammation (serum ALT, IU/L) in the same three patients with acute HBV and c) in two subjects experiencing spontaneous flares of chronic HBeAg− disease (days or months from initial presentation in clinic).