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. 2015 Jun 4;161(6):1453–1467. doi: 10.1016/j.cell.2015.04.053

Figure 4.

Figure 4

Comprehensive DNA Demethylation in hPGCs Revealed by Base-Resolution BS-Seq

(A) Violin plots showing distribution of CpG methylation levels in overlapped 1 kb genomic tiles of conventional H9 ESCs, Wk7 female gonadal somatic cells (soma), Wk5.5–Wk9 female and male hPGCs, sperm, and ICM. Common tiles with a minimum of 5 CpGs and at least 20% of the total CpGs covered by at least 5× in each sample are considered. These thresholds were applied to all subsequent methylation analyses unless stated otherwise. Due to low coverage, ICM only has ∼42% of common tiles fulfilling the above criteria. Black point indicates median.

(B) Averaged CpG methylation level profiles of all genes from 5 kb upstream (−) of transcription start sites (TSSs), through scaled gene bodies to 5 kb downstream (+) of transcription end sites (TES).

(C) Density plots illustrating DNA methylation dynamics of 1 kb tiles between indicated pairs of samples. Color intensity indicates tile counts within each bin, whereas blue regression lines show trends of methylation changes.

(D) K-means clustering of repeat-free tiles into seven dynamic groups. Gray tiles of ICM do not pass coverage thresholds and are therefore not shown. Tiles in cluster 6 retain partial methylation in hPGCs.

(E) Violin plots showing distribution of CpG methylation levels in human and mouse PGCs at conserved repeat-free 1 kb tiles.

(F) Unsupervised hierarchical clustering of methylation levels of conserved repeat-free 1 kb tiles in human and mouse PGCs.

See also Figure S4 and Table S2.