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. 2014 Sep 3;105(9):1152–1159. doi: 10.1111/cas.12479

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© 2014 The Authors. Cancer Science published by Wiley Publishing Asia Pty Ltd on behalf of Japanese Cancer Association.

This is an open access article under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Fig 2 — MiR-17 increases growth of synovial sarcoma cells. (a) Fuji and HS-SYII cells were infected with miR-17-producing or its control lentivirus, and the expression levels of miR-17 were examined by semi-quantitative RT-PCR. (b) Proliferation of Fuji and HS-SYII cells with or without MiR-17 overexpression was investigated. *P < 0.05 versus control cells. (c) Colony formation assay was performed in both Fuji and HS-SYII cells with or without miR-17 overexpression. The numbers and size were measured and graphed. ND means not detected. (d) Wound healing assay. Moved distances of Fuji and HS-SYII cells with or without miR-17 overexpression were measured at indicated time points. (e) Matrigel invasion assay. Invaded cells under the filter were counted and graphed.