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. 2015 May 27;3(5):e12344. doi: 10.14814/phy2.12344

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© 2015 The Authors. Physiological Reports published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society.

This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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The residual component of the AHP is mediated by K⁺ currents. (A) The reverse potential of the residual AHP is close to the calculated Ek. Empty circles represent the E_AHP of single neurons, whereas the black squares represent the average value of E_AHP ± SEM. (B) Removal of the extracellular K⁺ produced a transient increase, followed by a reduction, in the residual AHP. This specific pattern is possibly caused by the consecutive transient increase and sustained decrease in the potassium driving force due to the processes of extracellular and then intracellular potassium depletion. (C) The residual component of the AHP is reduced by the Ca²⁺ channel antagonists Ni²⁺/Cd²⁺. *Significant difference between the two conditions for each frequency. **P < 0.01.