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. Author manuscript; available in PMC: 2016 Jul 1.
Published in final edited form as: Atherosclerosis. 2015 Apr 26;241(1):69–78. doi: 10.1016/j.atherosclerosis.2015.04.805

Fig. 7. Increased expression of Grx2a in monocytes and macrophages did not affect HFD-induced atherosclerotic lesion formation.

Fig. 7

(A) Representative image of Oil red O (ORO)-stained sections of the aortic root from female and male LDLR−/− (wildtype; Wt) and transgenic Grx2aMacLDLR−/− mice (Tg) fed a HFD for 14 weeks. (B) Quantification of atherosclerosis in the aortic root wildtype and transgenic Grx2aMacLDLR−/− mice. There was no statistically significant difference in lesion size between LDLR−/− mice (open bars) and Grx2aMacLDLR−/− transgenic mice (closed bars). Females: n=9 per group; Males: n=12 per group.