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. 2015 Jun 1;11(7):845–859. doi: 10.7150/ijbs.11921

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Fig 3 — Rac1 was involved in bFGF-promoted dermal fibroblast migration under high glucose conditions. (A) Rac1-siRNA (10 nM) blocked bFGF-induced dermal fibroblast migration at 12 or 24 h after wounding, as detected through a wound-healing assay. (B, C) The migration rate of dermal fibroblast in the presence of Rac1 siRNA is expressed as the migrating distance per hour and analyzed. The data are presented as the means ± S.E.M. from six independent experiments. (D, F) bFGF enhanced the activity of Rac1, as detected by western blot. (E, G) Rac1-siRNA reduced the total Rac1 protein and the activity of Rac1 after bFGF treatment. *P < 0.05 and **P < 0.01 compared with the indicated group.

Fig 3 — Rac1 was involved in bFGF-promoted dermal fibroblast migration under high glucose conditions. (A) Rac1-siRNA (10 nM) blocked bFGF-induced dermal fibroblast migration at 12 or 24 h after wounding, as detected through a wound-healing assay. (B, C) The migration rate of dermal fibroblast in the presence of Rac1 siRNA is expressed as the migrating distance per hour and analyzed. The data are presented as the means ± S.E.M. from six independent experiments. (D, F) bFGF enhanced the activity of Rac1, as detected by western blot. (E, G) Rac1-siRNA reduced the total Rac1 protein and the activity of Rac1 after bFGF treatment. *P < 0.05 and **P < 0.01 compared with the indicated group.