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. 2015 Feb 4;6(7):5253–5262. doi: 10.18632/oncotarget.2867

Figure 1. Expression of human papillomavirus (HPV)-16E7, 6E7 and 16E7 mutants in primary human keratinocytes (PHKs).

Figure 1

(A) RT-PCR of HPV-16E7 in PHKs expressing 16E7 and 16E7 mutants L22A and C24G. β-actin was used as a control. (B) Western blot analysis of pRb and p130 protein in PHKs expressing 16E7 and 16E7 mutants L22A and C24G. GAPDH was a loading control. (C) Western blot analysis of pRb and p130 protein in PHKs expressing 6E7. Babe, vector control.