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. 2015 Jun 16;17(1):80. doi: 10.1186/s13058-015-0586-z

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© Pang et al.; licensee BioMed Central. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Copy number alterations of specific genes associated with ductal carcinoma in situ (DCIS) phenotype and disease stage. *Small DCIS lesions are defined as less than 15 mm, and large DCIS lesions are defined as more than 40 mm. aCGH, array-comparative genomic hybridization; CGH, comparative genomic hybridization; ER, estrogen receptor; FISH, fluorescence in situ hybridisation; HG, high grade; MLPA, multiplex ligation-dependent probe amplification; non-HG, non-high grade; PCR, polymerase chain reaction