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. 2015 Jun 20;16:86. doi: 10.1186/s12882-015-0075-8

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© Park et al. 2015

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Urinary AGT/Cr and PRA Levels According to CKD Stages and Hypertension. As a marker of intrarenal RAS activation, we measured urinary AGT/Cr and compared with PRA level, a marker of systemic RAS activation. Both urinary AGT/Cr and PRA were elevated in hypertensive subjects compared to normotensive subjects (P > 0.05). Among hypertensive subjects, urinary AGT/Cr was significantly increased in the advanced CKD stages (III-V, gray bar) compared to early CKD stages (I-II, white bar)(28.6 ± 60.3 vs. 93.2 ± 139.3 μg/g, P < 0.001). In contrast, PRA levels were not statistically different between CKD stage groups. AGT, angiotensinogen; CKD, chronic kidney disease; Cr, creatinine; PRA, plasma renin activity