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. 2015 Jul;22(7):336–343. doi: 10.1101/lm.038083.115

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© 2015 Venna et al.; Published by Cold Spring Harbor Laboratory Press

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Figure 3. — Western blot analysis confirmed the specificity and CNS availability of the GSK-3β inhibitor. (A) Representative Western blot images sham (sh) and stroke GSK-3β levels with vehicle and drug treatment (ss) (B) Quantification of GSK-3β levels show that at 6 h after stroke pGSK-3β levels increase with ischemia and this increase is significantly higher after inhibitor treatment. No significant changes in pLKB1 levels were detected. (*) P < 0.05; (n = 4 per sham group and n = 6 per stroke group).